Nine papers have been retracted from a confusing “sharing” operation at several hospitals

2022-05-13 0 By

Long non-coding RNA (lncRNA) HOX transcriptional antisense RNA (HOTAIR) has been found to be overexpressed and involved in tumor progression and metastasis in many human malignancies.Although the downstream target of HOTAIR regulating tumor metastasis is unclear, there is evidence that Mir-23b may be involved in this event.Zhu Zhenning, Shaanxi University of Traditional Chinese Medicine, Feb 8, 2018HOTAIR contributes to cell proliferation and Metastasis of cervical cancer viaTargeting Mir-23b /MAPK1 Axis “. This study identifies a novel pathway through which HOTAIR plays its carcinogenic role and provides a molecular basis for its potential application in the prognosis and treatment of cervical cancer.However, on June 8, 2021, this article was withdrawn, mainly because the experimental samples, rulers and tablecloths of this article were reused in as many as 10 other articles, and the author did not respond to the editor’s inquiries.The author’s employer was Shaanxi University of Chinese Medicine, but it was spelt Shanxi University of Chinese Medicine.Upon notification from the reader and at the request of the editor and editorial board, this article will be withdrawn from Bioscience Reports to draw the editorial board’s attention to Figures 2C, 3C, 5E, 6J and 7A, which contain overlapping data and images that duplicate with several other publications.The author was contacted about the retraction, but did not respond to the magazine’s inquiries and concerns.In view of the seriousness of the issues raised, the editorial board supported the decision to withdraw the article.The Retraction editorial board found that the tablecloth used in the article was highly similar to that used by the authors of 10 other articles in different units, as follows:[1] Shaanxi University of Traditional Chinese Medicine, Bioscience Reports, Figure 7A[2] Wuhan University, ONCOLOGY LETTERS, Figure 4A[3] Taizhou Municipal Hospital, Zhejiang Province, ONCOLOGY Research, Figure 6A[4] General Hospital of The Chinese People’s Liberation Army Chengdu Military Region,[6] International Journal of Oncology, FIG. 8A[5] Yulin Hospital of Traditional Chinese Medicine, Oncology Reports, FIG. 6A[6]Figure 6A[7] China Medical University, Oncology Research, Figure 7A[8] Shandong Liaocheng People’s Hospital, Oncology Research, Figure 6A[9] Lanzhou University, Oncology Research, Figure 7A[10] Tongji University, ChinaBiochemical and Biophysical Research Communications, FIG. 4A[11]The Retraction editorial board has also found that the sample used for this article overlaps with the sample/curve used for other articles:[1]HOTAIR contributes to cell proliferation and metastasis of cervical cancer via targetting /MAPK1 Axis (rejected) [2]Inhibition of BAMBI − − reducing the penalty and motility of colon cancervia activating TGF‑β/Smad pathway in cancerRole of Epigenetic silence in vitro and in vivo (rejected) [3]Epigenetic silence of HOTAIR contributes to the metastasis of pancreatic ancer via targetingMir-138 (dismissed) [4]The inhibitory effect of MEG3/ Mir-214 /AIFM2 axis on The growth of T-cell lymphoblasticLymphoma (Published in Chinese) [5]Long non-coding RNA MEG3 inhibits cell growth of Gliomas by targeting Mir-93 and inactivating PI3K/AKTPathway (Deleted) [6]HNF1A AS1 Promotes growth and metastasis of SQUamous cell carcinoma by sponging Mir-214 toUpregulate the expression of sox-4 (withdrawn) [7]UCA1 Regulates the Growth and Metastasis of Pancreatic Cancer bySpongingMir-135a (Withdrawn) [8]Silencing of lncRNA CCDC26 Restrains the Growth and Migration of Glioma Cells In Vitroand In Vivo viaTargeting miR-203[9] Long Noncoding RNA SChLAP1 Accelerates the Proliferation and Metastasis of Prostate Cancervia Targeting Mir-198 and Promoting the MAPK1 Pathway [10]Astragaloside IV inhibits progression of glioma via blockingMAPK/ ERKsignaling pathway[11]H19 promotes the migration and invasion of colon cancer by spongingmiR-138 to upregulate The Expression of HMGA1 this article is based on the Retraction public account